The potential for polycyclic aromatic hydrocarbons (PAHs) to have adverse effects that persist across generations is an emerging concern for human and wildlife health. This study evaluated the role of mitochondria which are maternally inherited in the cross-generational toxicity of benzo(a)pyrene (BaP) a model PAH and known mitochondrial toxicant. Mature female zebrafish (F0) were fed diets containing 0 12.5 125 or 1250 μg BaP/g at a feed rate of 1% body weight twice/day for 21 days. These females were bred with unexposed males and the embryos (F1) were collected for subsequent analyses. Data herein include mitochondrial function of organs ex vivo (brain heart liver and ovary) from exposed adults as well as mitochondrial function raw data and profiles for their F1 embryos.
